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From the Townsend Letter
October 2006


Recent Progress in Clinical Applications
and Research in Fibromyalgia

by Robert W. Bradford, Professor of Medicine, D. Sc. NMD, and Henry W. Allen, Director of Clinical Biochemistry, BRI

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Live Cell Injections
The intermuscular injection of fetal cells in FMS patients has improved energy levels, reduced pain, decreased depression, and improved neurological function in all patients.60

The results of this open clinical outcome study clearly indicate that a broad-base antimicrobial protocol (AMP) will significantly reduce the persistent pain and muscle weakness in the FMS patient (95.2%). Although we are able to observe the various fungal forms (white, black, red, brown) by High Resolution Microscopy as well as correlate (from the literature) the symptoms of the fungal infection with the symptoms of FMS, additional research is needed to define the specific species of fungi as well as determine the role of mycoplasma, virus, and bacteria also observed in High Resolution Blood Morphology (HRBM™).

The early FMS patients had a high reoccurrence of the FMS symptoms within 30 days of the initial AMP. This reoccurrence was determined to be a result of reinfection of the systemic system from two primary sources. First, there were focal infections in the intestinal tract resulting in reinfection of the systemic system. This problem was reduced significantly by adding oral antifungal medication to the post-therapy protocol.

Secondly, it was observed that quite a few of the male spouses of the FMS patients showed the same high concentration of fungal forms and mycoplasma in the blood as the patients. These male spouses had some secondary symptoms of a minor nature (but without the classical muscle pain and weakness), yet they were reinfecting their female spouses. By treating the infected male spouses with the AMP, there was a significant reduction in the reoccurrence of FMS. These observations only confirm the basic premise based on microscopic evidence, that FMS has a microbial cause, further confirmed by the reduction in symptoms from antimicrobial therapy. See Chart 14.

Chart 14: Risk Factors for Reoccurrence

A. A localized infection in the intestinal tract.
B. Infected spouse carrier.
C. A deficient immune system.
D. Environmental factors - Sick building syndrome.
E. Lyme Disease

In summary, FMS represents a major burden in our society. Between three to six million infected patients suffer from muscle pain and weakness, fatigue, depression, heart abnormalities, and bowel dysfunction with numerous other secondary effects. There is no approved treatment, and traditional therapies have not resolved FMS. This limited open prospective study clearly demonstrates the hypothesis that the etiology of fibromyalgia is infectious microorganisms, predominantly fungal with possible synergistic infection by mycoplasma, virus, and bacteria. A variety of risk factors make reoccurrence of the disease a very real possibility. Carefully planned studies can be designed to specifically investigate the role of infectious organisms, toxicity states, and functional imbalances. The results of such studies will undoubtedly shed more light on our attempts to resolve this most perplexing disease.



For comments or more information, you may reach Dr. Bradford at 888-339-9355 or by email at

The authors wish to acknowledge the valuable contribution and meaningful input of
Prof. Ann Marie Dixon, NMD, Albert MacKenzie, MD, Los Angeles, California, Norbert Becquet, MD, Little Rock, Arkansas, and the staff of Ingles Integrative Hospital, Tijuana, BC, Mexico.

Neither this monograph nor any part may be reproduced or transmitted in any form or by any means, electronic or mechanical, including photocopying, microfilming, and recording, or by any information storage and retrieval system, without permission in writing from the authors.

© 2002/2006 Bradford Research Institute, Chula Vista, California 91911
The Bradford Research Institute has published over 75 research papers and five textbooks on the etiology, biochemistry and therapeutic implications of disease processes and their functional implications.

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